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Fine-sampled Photographic Quantitation of Dermal Wound Healing Senescence in Aged BALB/cByJ Mice and Therapeutic Intervention with FGF-1
Mathematical Model for the Determination of Mouse Excisional Wound Healing Parameters from Photographic Data
Investigating the Dynamics and Polyanion Binding Sites of Fibroblast Growth Factor-1 Using Hydrogen-Deuterium Exchange Mass Spectrometry
Folding Nucleus Structure Persists in Thermally-Aggregated FGF-1
9783587
Small animal restraining harness or jacket
Fibroblast growth factor mutants having improved functional half-life and methods of their use
S116R Phosphorylation Site Mutation in Human FGF-1 Differentially Affects Mitogenic and Glucose Lowering Activities
Myths and Verities in Protein Folding Theories
Evolution of a protein folding nucleus.
Engineering a Cysteine-Free Form of Human Fibroblast Growth Factor-1 for "Second Generation" Therapeutic Application.
Engineering a Cysteine-Free Form of Human Fibroblast Growth Factor-1 for "2nd Generation" Therapeutic Application
Evolution of a Protein Folding Nucleus
Accelerated healing in NONcNZO10/LtJ type 2 diabetic mice by FGF 1
Fibroblast growth factor mutants having improved functional half-life and methods of their use
single aromatic core mutation converts a designed "primitive" protein from halophile to mesophile folding.
Single Aromatic Core Mutation Converts a Designed "Primitive" Protein from Halophile to Mesophile Folding
Mutation Choice to Eliminate Buried Free Cysteines in Protein Therapeutics
Evolution and design of protein structure by folding nucleus symmetric expansion
SYMMETRIC PROTEIN ARCHITECTURE IN PROTEIN DESIGN: TOP-DOWN SYMMETRIC DECONSTRUCTION
Prebiotic Protein Design supports a Halophile Origin of Foldable Proteins
Prebiotic protein design supports a halophile origin of foldable proteins.
Isomannide-based peptidomimetics as inhibitors for human tissue kallikreins 5 and 7.
Kallikrein cascades in traumatic spinal cord injury
Kallikrein 6 Signals through PAR1 and PAR2 to Promote Neuron Injury and Exacerbate Glutamate Neurotoxicity
Alternative Folding Nuclei Definitions Facilitate the Evolution of a Symmetric Protein Fold from a Smaller Peptide Motif
Kallikrein-related Peptidase 6
Fibroblast growth factor mutants having improved functional half-life and methods of their use
Simplified protein design biased for prebiotic amino acids yields a foldable, halophilic protein.
Activation profiles of human kallikrein-related peptidases by matrix metalloproteinases.
Activation Profiles of Human Kallikrein-Related Peptidases by Matrix          Metalloproteinases
Simplified Protein Design Biased for Pre-Biotic Amino Acids Yields a Foldable, Halophilic Protein
Experimental support for the foldability-function tradeoff hypothesis
Emergence of Symmetric Protein Architecture from a Simple Peptide Motif
Mutants of human fibroblast growth factor having increased stability and/or mitogenic potency
Mutants of human fibroblast growth factor having increased stability and/or mitogenic potency
Mutants of human fibroblast growth factor having increased stability and/or mitogenic potency
Kallikrein 6 is a Novel Molecular Trigger of Reactive Astrogliosis
Pharmacokinetic properties of 2nd-generation fibroblast growth factor-1 mutants for therapeutic application.
Pharmacokinetic Properties of 2(nd)-Generation Fibroblast Growth Factor-1 Mutants for          Therapeutic Application
Experimental support for the evolution of symmetric protein architecture from a simple peptide motif.
Experimental Support for the Evolution of Symmetric Protein Architecture from a Simple          Peptide Motif
Expression and Function of the Kallikrein-Related Peptidase 6 in the Human Melanoma          Microenvironment
Functional Role of Kallikrein 6 in Regulating Immune Cell Survival
Engineered human acidic fibroblast growth factors and associated methods
Mutant polypeptides of fibroblast growth factor 1
Method of treating multiple sclerosis with anti-K6 antibody
Mutant polypeptides of fibroblast growth factor 1
Functional intersection of the kallikrein-related peptidases (KLKs) and thrombostasis axis.
Mutants of human fibroblast growth factor having increased stability and/or mitogenic potency

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