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Accelerated healing in NONcNZO10/LtJ type 2 diabetic mice by FGF 1
Experimental support for the evolution of symmetric protein architecture from a simple peptide motif.
Prebiotic protein design supports a halophile origin of foldable proteins.
SYMMETRIC PROTEIN ARCHITECTURE IN PROTEIN DESIGN: TOP-DOWN SYMMETRIC DECONSTRUCTION
Experimental Support for the Evolution of Symmetric Protein Architecture from a Simple          Peptide Motif
Prebiotic Protein Design supports a Halophile Origin of Foldable Proteins
X-ray structure and biophysical properties of rabbit fibroblast growth factor 1.
Autolytic Regulation of Human Kallikrein-Related Peptidase 6
Functional Intersection of the Kallikrein-Related Peptidases (KLKs) and Thrombostasis          Axis
Activation Profiles of Human Kallikrein-Related Peptidases by Matrix          Metalloproteinases
X-ray Structure and Biophysical Properties of Rabbit Fibroblast Growth Factor 1
Kallikrein-related Peptidase 6
Pharmacokinetic Properties of 2(nd)-Generation Fibroblast Growth Factor-1 Mutants for          Therapeutic Application
Simplified protein design biased for prebiotic amino acids yields a foldable, halophilic protein.
Experimental support for the foldability-function tradeoff hypothesis
Simplified Protein Design Biased for Pre-Biotic Amino Acids Yields a Foldable, Halophilic Protein
Mutation Choice to Eliminate Buried Free Cysteines in Protein Therapeutics
Emergence of Symmetric Protein Architecture from a Simple Peptide Motif
Pharmacokinetic properties of 2nd-generation fibroblast growth factor-1 mutants for therapeutic application.
Activation profiles of human kallikrein-related peptidases by matrix metalloproteinases.
Functional intersection of the kallikrein-related peptidases (KLKs) and thrombostasis axis.
Engineering an improved crystal contact across a solvent-mediated interface of human fibroblast growth factor 1.
autolytic regulation of human kallikrein-related peptidase 6.
Completed KLK Activome Profile
Crystal Structure and Biochemical Characterization of Human Kallikrein 6 Reveals a Trypsin-like Kallikrein is Expressed in the Central Nervous System
Engineering an Improved Crystal Contact Across a Solvent-Mediated Interface of Human          Fibroblast Growth Factor 1
Kallikrein 6 is a Novel Molecular Trigger of Reactive Astrogliosis
S(1)' and S(2)' Subsite Specificities of Human Plasma Kallikrein and Tissue Kallikrein          1 for the Hydrolysis of Peptides Derived from the Bradykinin Domain of Human          Kininogen
Activation Profiles of Human Kallikrein-Related Peptidases by Proteases of the          Thrombostasis Axis
Protease-Activated Receptor Dependent and Independent Signaling by Kallikreins 1 and 6          in CNS Neuron and Astroglial Cell Lines
Engineering a Cysteine-Free Form of Human Fibroblast Growth Factor-1 for "2nd Generation" Therapeutic Application
Kallikrein 6 Signals through PAR1 and PAR2 to Promote Neuron Injury and Exacerbate Glutamate Neurotoxicity
Expression and Function of the Kallikrein-Related Peptidase 6 in the Human Melanoma          Microenvironment
Evolution of a protein folding nucleus.
Alternative Folding Nuclei Definitions Facilitate the Evolution of a Symmetric Protein Fold from a Smaller Peptide Motif
Single Aromatic Core Mutation Converts a Designed "Primitive" Protein from Halophile to Mesophile Folding
Evolution of a Protein Folding Nucleus
Evolution and design of protein structure by folding nucleus symmetric expansion
Molecular Modeling of Substrate Binding in Wild-type and Mutant Corynebacteria 2,5-diketo-D-gluconate Reductases
Investigating the Dynamics and Polyanion Binding Sites of Fibroblast Growth Factor-1 Using Hydrogen-Deuterium Exchange Mass Spectrometry
Engineering a Cysteine-Free Form of Human Fibroblast Growth Factor-1 for "Second Generation" Therapeutic Application.
Kallikrein cascades in traumatic spinal cord injury
Substrate Specificity of Human Kallikreins 1 and 6 Determined by Phage Display
Kallikreins are Associated with Secondary Progressive Multiple Sclerosis and Promote          Neurodegeneration
Functional Role of Kallikrein 6 in Regulating Immune Cell Survival
S116R Phosphorylation Site Mutation in Human FGF-1 Differentially Affects Mitogenic and Glucose Lowering Activities
Folding Nucleus Structure Persists in Thermally-Aggregated FGF-1
single aromatic core mutation converts a designed "primitive" protein from halophile to mesophile folding.
Mutagenesis of the Crystal Contact of Acidic Fibroblast Growth Factor
Mathematical Model for the Determination of Mouse Excisional Wound Healing Parameters from Photographic Data

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